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 Anticonvulsants induce some metabolic pathways suitable to docetaxel. CYP450 and CYP3A display greater expression in response to using anticonvulsants and the metabolism of docetaxel metabolite M4 is processed by these CYPs. A corresponding increase in clearance of M4 by 25% is observed in sufferers getting phenytoin and phenobarbital, common anticonvulsants.[twelve] Prevalent and/or very likely drug-drug combinations and known Unintended effects from drug interactions
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